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MessagePosté le: Sam 3 Sep - 06:57 (2016)    Sujet du message: Mitochondrial Theory Of Aging Pdf Download Répondre en citant




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or its licensors or contributors. The classical pathway is indicated with blue arrows; the generation of ROS (superoxide anion, hydrogen peroxide, and hydroxyl radicals), as a by-product of mitochondrial oxidative phosphorylation, results in damage to mitochondrial macromolecules including the mtDNA, the latter leading to deleterious mutations (3). This makes the respiratory chain less efficient, and physiologically is used for thermogenesis in brown fat. Once a DSB has formed, repair of the mtDNA molecule will be attempted by exonuclease activity which initially creates ssDNA. & Glick, E. Recent physicochemical modeling suggests that once formed these deleted mtDNA species have inherent stability [27].

Chinnery, Wellcome Trust Centre for Mitochondrial Research, Institute of Genetic Medicine, Newcastle University, UKReceived 19 March 2015, Revised 21 May 2015, Accepted 26 May 2015, Available online 4 June 2015Highlights•Mitochondrial dysfunction retains a central position in aging theory.•Clonal expansion of mtDNA mutations occurs in aging post-mitotic tissues.•ROS may have both deleterious and beneficial roles.AbstractThe free radical theory of aging is almost 60years old. 5 , 741747. Related Article Biological Sciences - Cell Biology: Aleksandra Trifunovic, Anna Hansson, Anna Wredenberg, Anja T. D., Someya, S., Panzer, K., Wohlgemuth, S. analyzed data and wrote the paper. (2005) Science 308 , 19091911. These mice have a homozygous knock-in mutation (PolgD257A/D275A) for an error-prone polymerase gamma (the sole mtDNA polymerase). Some suggestions: Go back to the last page Go to the home page .. Do the mitochondrial mutations introduced by Pol- result in mutant mitochondrial proteins that are defective in coupling of oxygen metabolism with ATP causing increased ROS production, DNA damage, and mutations? This scenario would be consistent with the venerable free radical theory of aging (8).

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